Toxic epidermal necrosis
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A cikin sakamakon Stiftung Warentest na 2022 daga Jamus, gamsuwar mabukaci tare da ModelDerm ya ɗan yi ƙasa kaɗan fiye da biyan shawarwarin telemedicine.
A cikin sakamakon Stiftung Warentest na 2022 daga Jamus, gamsuwar mabukaci tare da ModelDerm ya ɗan yi ƙasa kaɗan fiye da biyan shawarwarin telemedicine.
Halayen asarar fata na cutar Toxic epidermal necrosis
relevance score : -100.0%
ReferencesStevens-Johnson Syndrome (SJS) da Toxic Epidermal Necrolysis (TEN) yanayi ne da ba kasafai ake samu ba inda fata ke fuskantar babban necrosis da zubarwa. Dangane da jiyya, cyclosporine yana da tasiri sosai ga SJS, yayin da haɗin immunoglobulin na ciki (IVIg) da corticosteroids ke ba da sakamako mafi kyau a lokutan SJS da TEN.
Stevens-Johnson Syndrome (SJS) and Toxic Epidermal Necrolysis (TEN) are rare diseases that are characterized by widespread epidermal necrosis and sloughing of skin. Regarding treatment, cyclosporine is the most effective therapy for the treatment of SJS, and a combination of intravenous immunoglobulin (IVIg) and corticosteroids is most effective for SJS/TEN overlap and TEN.
Stevens-Johnson Syndrome (SJS) and Toxic Epidermal Necrolysis (TEN) are rare diseases that are characterized by widespread epidermal necrosis and sloughing of skin. Regarding treatment, cyclosporine is the most effective therapy for the treatment of SJS, and a combination of intravenous immunoglobulin (IVIg) and corticosteroids is most effective for SJS/TEN overlap and TEN.
Toxic epidermal necrolysis (TEN) cuta ce mai muni wadda ke haifar da lalacewar epidermis, tare da raguwa mai yawa daga saman fata, wanda ke shafar sama da kashi 30 % na jikin mutum. TEN na da adadin mace‑mace sama da 20 %, sau da yawa saboda cututtuka da wahalar numfashi. Dakatar da maganin da ke haifar da amsawa, samar da kulawar tallafi, da amfani da ƙarin jiyya na iya inganta sakamakon. Nazarin kwanan nan ya nuna cewa kwayoyi kamar cyclosporine, tumor necrosis factor‑alpha inhibitors, intravenous immunoglobulin (IVIG) da corticosteroids na iya taimakawa, bisa ga gwaje‑gwajen da aka gudanar da kuma nazarin bincike da yawa.
Toxic epidermal necrolysis (TEN) is a serious skin reaction caused by certain medications and immune system activity, resulting in large-scale detachment of the outer skin layer (epidermis), affecting more than 30% of the body's surface. TEN has a mortality rate of over 20%, often due to infections and breathing difficulties. Stopping the medication causing the reaction, providing supportive care, and using additional treatments can improve the outcome. Recent studies have shown that drugs like cyclosporine, tumor necrosis factor alpha inhibitors, and a combination of intravenous immune globulin and corticosteroids can be helpful, based on randomized controlled trials and analyses of multiple studies.
Toxic epidermal necrolysis (TEN) is a serious skin reaction caused by certain medications and immune system activity, resulting in large-scale detachment of the outer skin layer (epidermis), affecting more than 30% of the body's surface. TEN has a mortality rate of over 20%, often due to infections and breathing difficulties. Stopping the medication causing the reaction, providing supportive care, and using additional treatments can improve the outcome. Recent studies have shown that drugs like cyclosporine, tumor necrosis factor alpha inhibitors, and a combination of intravenous immune globulin and corticosteroids can be helpful, based on randomized controlled trials and analyses of multiple studies.
Recent Advances: There is improved understanding of pain and morbidity with regard to the type and frequency of dressing changes. More modern dressings, such as nanocrystalline, are currently favored as they may be kept in situ for longer periods. The most recent evidence on systemic agents, such as corticosteroids and cyclosporine, and novel treatments, are also discussed.
Dalilan da suka fi dacewa su ne wasu magunguna irin su lamotrigine, carbamazepine, allopurinol, sulfonamide (maganin rigakafi), da nevirapine. Abubuwan haɗari sun haɗa da HIV da lupus erythematosus. Jiyya yawanci ana yi a asibiti, kamar a sashin ƙonawa ko sashin kulawa mai zurfi.
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Wannan cuta ce mai tsanani, don haka idan laɓɓanka ko bakinka sun kamu, ko fatar jikinka ta yi kumbura, ka garzaya ga likitanka da wuri‑wuri.
Ya kamata a daina amfani da magungunan da ake zargi da haifar da cutar. (misali maganin rigakafi, magungunan anti‑inflammatory marasa steroidal)